Methylation, Pregnancy Loss and Nutrient Restoration
Claire was 38 and had experienced three early pregnancy losses after conceiving her first child easily at 32. Every standard investigation came back normal. She had been diagnosed with unexplained recurrent pregnancy loss and told there was nothing more that could be done.
Her blood work revealed severe B12 depletion and elevated homocysteine. Genetic testing confirmed a double MTHFR mutation that reduced her methylation capacity by 70%. She had been taking a standard prenatal with synthetic folic acid, which her body could not convert. The very supplement designed to support her pregnancy was blocking the pathway she needed most.